Each spring, the Cinema Pacific Film Festival hosts a special event called the Adrenaline Film Project (AFP). Working in teams, students and local filmmakers are given just seventy-two hours to write, shoot, and edit a short film from start to finish. During this intensive film production workshop, visiting industry professionals (such as Jeff Wadlow, Leigh Kilton-Smith, and Omar Naim) monitor the participants over the three-day extravaganza.
At the end of last month, I had the opportunity to attend the final screening of the 2014 Adrenaline Film Project. Every year, Cinema Pacific chooses a region (or two) around the world as the focus of the festival. Required props for AFP are connected in some way to the specific region, as well as the required line of dialogue that each team must incorporate. This year, AFP focused on Taiwan and Chile. For the Taiwanese prop, each team was required to incorporate a red teapot into each of their films. The Chilean line of dialogue was “So, you think you’re death?” meaning, “you think you’re all that.”
Within the twelve films produced by teams of three participants, each of the following genres was represented (assigned randomly to each team): workplace comedy, dark comedy, B-movie, coming of age, horror, drama, noir, revenge, psychological thriller, sci-fi, heist, and romantic comedy.
After each film was shown, the audience voted for their favorite film. I appreciated the creativity of the voting process, as you actually use the program handed to you upon entrance as your ballot. The winner of the Audience Award was the dark comedy, “Kill for Her” directed by Will Cuddy, Zachary Feiner, and Tommy Pittenger.
Other awards given throughout the night were the Mentor Award and the Best Actor Award. The mentor award is awarded by the mentors, of course, and was given to the revenge film, “Get Your Fill” by Elijah Sprints, Colin Zeal, and Talon Isak Sherer. The Best Actor Award went to Kim Fairbairn from the psychological thriller, “Bottom Line.”
The last award, The Ben Kalb Jury Award, is given to the team who demonstrates the strongest overall production and the most faithful and accurate execution of their assigned genre. Every year, the awarding of this prize honors the contributions of Ben Kalb, whose generous sponsorship makes the UO AFP possible. During this screening, Ben Kalb was in attendance and gave a speech, announcing that he plans on donating his financial support for at least five more years. The winner of the Ben Kalb Jury Award was the heist film, “Taking Tibet” by Derek Brown, Jacob Salzberg, and Noah Phillips-Edwards.
I first heard about AFP in the same room where our class now meets, Mackenzie 214, during an English 260 course that I took in my freshman year. I was able to pull a few friends together to participate in both the 2012 and 2013 projects. This grueling, challenging, and satisfying endeavor has taught me that the final product on the big screen is only a small portion of the entire production. Sadly, they created new rule where you can only participate two years overall, but I definitely plan on attending the screening next year as well!
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This recap of the Adrenaline Film Project really captures the excitement and creativity that such intense film challenges bring out in participants. I love how the festival not only pushes filmmakers to work under pressure but also ties in cultural elements like the Taiwanese red teapot and the Chilean dialogue line. It’s impressive how much variety the twelve films covered, showing the versatility of the teams in tackling different genres.
The voting system sounds very interactive and engaging—using the entrance program as a ballot is such a clever way to get the audience involved. Also, it’s inspiring to hear about Ben Kalb’s ongoing support, which clearly helps keep this unique event thriving year after year.
Reading about this reminded me of how digital initiatives like eDistrict UP aim to empower local communities by integrating culture and technology. Just like AFP nurtures young filmmakers and creativity, eDistrict UP focuses on connecting people through innovation and accessibility.
Thanks for sharing your experience and insights! It really highlights the value of collaboration and creativity in both film and community development.
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Ultimately, gene transcription may be modulated by these ‘non-genomic’ pathways but
a well-recognized exception is the fast elevation of calcium ion influx by a pathway
that’s confined to the cytoplasm. It isn’t
currently known whether non-genomic actions of androgens at
physiological concentrations are necessary in skeletal muscle development,
let alone what the non-genomic effects could also be evoked by the administration of
Anabolic steroids and bone density steroids.
For the sake of brevity, this review will only very briefly touch once more on non-genomic pathways beneath
‘Behavioural Effects’ (see the section ‘Behavioural mechanisms’).
The systematic doping program of the previous German Democratic Republic (GDR) provided
evidence of anabolic steroid and amphetamine use in female athletes (Karila, Kerlan, Christin-Maitre, 2024).
In the 1970 s, GDR athletes were given anabolic steroids
to enhance efficiency. Their dominance on the 1976 Montreal Olympics, winning eleven of thirteen swimming occasions, raised suspicions as a end result of atypical physical traits like deep
voices and broad shoulders, typical signs of steroid-induced masculinization.
This is partly facilitated by the co-localization of the AR and β-catenin within the
cell nucleus; additional, β-catenin preferentially binds
to the AR over a number of different steroid hormone receptors including these of estrogen, progesterone, and glucocorticoids (22).
B-catenin’s function as a coactivator of the AR causes a forced overexpression of β-catenin to augment AR-mediated transcription (22).
This signaling by way of the AR has also been noticed
to interact with Notch signaling (22).
Testosterone is produced instantly from the ovaries and also through conversion of weaker androgens,
androstenedione and dehydroepiandrosterone
(DHEA), which are synthesized by the ovaries and the adrenal glands, respectively.
In young premenopausal women, the ovary is answerable for approximately 25% of the testosterone production, whereas 75% is
derived from the adrenal androgens. Nonetheless, in postmenopausal ladies, the ovary becomes a significant source of testosterone manufacturing and its contribution will increase to 50% (Adashi,
1994). Although the climacteric ovary is atrophic and loses its capacity to
synthesize estradiol and follicles, it continues to secrete androgens under the stimulation of postmenopausal gonadotropins (Sluijmer et al.,
1995; Dowsett et al., 1980; Dennefors et al., 1980).
AAS addiction is generally thought-about to be a psychic dependancy, but
the withdrawal effects that happen when AAS use stops clearly indicate
an element of bodily habit as well.
AAS bind to androgen receptors, selling muscle development by increasing protein synthesis
and selling the expansion hormone-insulin-like progress factor
1 axis. Injectable forms of AAS have extended half-life due
to esterification, while oral AAS face decreased bioavailability because of first-pass metabolism.
Opposed effects of AAS embrace cardiovascular,
endocrine, neuropsychiatric and dermatological issues.
Anabolic androgenic steroids intake can lead to suppressing gonadotropins, resulting in reduced sperm focus, testicular dysfunction and hypogonadism, which may result in decreased
libido and erectile dysfunction. AAS use is widespread as a result of their capability
to enhance muscle growth for aesthetic functions and athletes’ performance, minimizing androgenic
effects. AAS use is very in style and 1–3% of US
inhabitants have been estimated to be AAS customers.
The use of anabolic steroids is either forbidden or closely managed in most human and some equine sports.
In contrast with the antagonistic effect on LDL- and HDL-cholesterol, AAS appear
to exert a impartial or favorable impact on lipoprotein (a)
(Lp(a)). Elevated Lp(a) levels are thought-about a longtime causal danger
factor for CVD (140). In a double-blind trial, nandrolone decanoate (200 mg weekly) for 8 weeks decreased Lp(a) compared with baseline, but not compared with placebo, in a bunch of bodybuilders (124).
In the same publication, a second nonblinded
trial is described during which AAS users self-administer their own cycle.
Herein, Lp(a) plummeted (-83%) compared with baseline
after 8 weeks, with partial return to baseline 6 weeks after cessation of AAS use.
Nonetheless, 600 mg testosterone enanthate weekly for
three weeks did not change Lp(a) in elderly obese men (119), but 200 mg testosterone enanthate weekly for 3 weeks decreased
Lp(a) substantially by 38% compared with baseline
in healthy male weightlifters – though a placebo group
was missing (141).
If the testosterone is very high however the FSH and LH ranges are low,
this provides proof that the individual is utilizing anabolic steroids.
AAS-induced hepatotoxicity was hypothesized to be associated to oxidative
stress in hepatic cells. Following AR receptor activation a rise
in reactive oxygen species can be noticed as a result of increase in mitochondrial
b-oxidation. Furthermore, antioxidant substances have a protecting position towards hepatotoxicity mediated by AASs.
It was also demonstrated that androgenic efficiency and metabolic resistance are positively linked to the degree of liver
harm [82].
Different short-acting testosterone preparations include
those which may be designed to be administered by the sublingual
or buccal route. Such short-acting formulations are of specific
concern in sport, as the exogenous supply of testosterone is rapidly eliminated following cessation of treatment.
Increased out-of-competition testing helps to fight the cheat who’s
using short-acting preparations and ceasing administration prior to
competitors in anticipation of testing. It is of interest that
an illicit preparation referred to as ‘The Cream’ was designed for transdermal utility (see the section ‘Designer steroids’).
This is a creating area and the comparative significance
of many of those coregulators is yet to be established for any
specific cell kind, not to mention their relative in vivo importance in examining tissue differences in androgen action. It is envisaged that genetic manipulation of the mouse will assist in elucidating their physiological relevance.
Testosterone is irreversibly transformed by the enzyme 5α-reductase to 5α-dihydrotestosterone (DHT), which binds with greater affinity to the androgen receptor (AR), or by aromatase to oestradiol, which binds to the oestrogen receptor (ER).
Their deleterious impact is further augmented by the fact, that such tumors have
a high incidence in younger folks, the cohort of people abusing
AAS. Additional studies are necessary to investigate the potential
hyperlink between AAS abuse and most cancers.
AASs are a known cause numerous functional issues, which can have an effect on liver, cardiovascular,
reproductive, musculoskeletal, endocrine, renal, immunologic
or neuropsychiatric techniques. Beyond these deleterious
macro-effects, testosterone-derived androgens could act instantly on mobile capabilities, with either genetic or epigenetic factors figuring out toxic, mutagen, genotoxic
and carcinogenic results. AAS also can affect most cancers
cell proliferation through genomic and non-genomic mechanisms, such
as the so-called estrogen-dependence mediated by ER, aromatase expression and IGF-1 manufacturing, which can even amplify each other.
Due To This Fact, IGF-1 could be considered as a
well-known most cancers inducer and promoter affecting
each stage of tumor growth, from cellular proliferation to the metastatic section.
For this cause, they’re reserved just for moderate
to severe inflammatory conditions (34). A widespread psychological side effect of AAS use is
muscle dysmorphia, in which users become preoccupied with having a muscular physique (31).
The only approach to obtain and use AAS legally would be to have them prescribed by a medical
skilled for a certain situation, corresponding to
low testosterone or a muscle-wasting illness.
This misunderstanding is partially perpetuated by the common but non-scientific time
period, “‘roid rage,” which could appear to recommend that
AAS customers display abrupt outbursts of temper or
sudden acts of violence. Although such behaviors may happen, it is extra
frequent that AAS users report a “slow boil” somewhat than a
sudden outburst. More specifically, they frequently
describe continual obsessional preoccupations that they cannot get out of their heads – similar to Mr.
A’s fixation on left-wing “media bias” or Mr.
B’s obsession with getting money to buy throws.
Other examples embrace one AAS person who described an instance the place he ordered pancakes and specified that
he didn’t need butter. He reported that for days afterwards,
he found himself continually preoccupied with this error, even waking up in the midst of the evening and angrily thinking “but why did she bring me butter once I had particularly explained that I didn’t need butter?
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